Regular garlic consumption can help lower cholesterol, reduce blood pressure, and prevent clots — all heart disease risk factors. Research shows garlic can influence estrogen levels in the body, perhaps helping reduce age-related bone loss.
Garlic. Garlic is part of the onion family and is a source of isoflavonoids, containing about 603mcg of phytoestrogens per 100g. Garlic is used in cooking throughout the world and therefore offers a simple way to include an estrogen rich food in your diet.
Medications used as estrogen blockers include: Aromatase inhibitors (AI): This medication can increase testosterone, along with other hormones, by inhibiting the ability of androgens to be converted to estradiol in the body. This decreases the overall level of estrogen in the body.
DIM (Diindolylmethane/3,3′-Diindolylmethane) is among the most effective estrogen blockers found in nature. It occurs naturally in cruciferous plants such as broccoli, cauliflower, Brussels sprouts and kale.
Packed within cruciferous veggies are phytochemicals that block the production of estrogen, allowing them to be an effective addition to an anti-estrogen diet. This group of vegetables includes kale, broccoli, cauliflower, Brussels sprouts, and arugula.
Large amounts of turmeric might interfere with the effects of estrogen. Taking turmeric along with estrogen might decrease the effects of estrogens. Some estrogen pills include conjugated equine estrogens (Premarin), ethinyl estradiol, estradiol, and others.
Garlic supplementation increases testicular testosterone and decreases plasma corticosterone in rats fed a high protein diet.
Blackberries, blueberries, cranberries, raspberries, and strawberries can all help to increase estrogen levels. As well as being rich sources of fiber, vitamins, and antioxidants, berries contain high levels of phytoestrogen lignans.
Including an array of fresh (or dried) herbs and spices especially those that have anti-inflammatory properties such as ginger, turmeric, sumac, paprika, garlic will promote hormone harmony as well as ensuring we are adding an array of different nutrients to our meals.
Cruciferous vegetables.
Broccoli, cauliflower, kale, collard greens, brussels sprouts, turnips, arugula and all the other wonderful, sulfur-rich foods in this plant family contain 3,3'-diindolymethane (DIM). DIM is chemoprotective, helps reduce high estrogen levels and supports phase 1 of estrogen detox in the liver.
Red grapes are another estrogen-blocking food that men should consider consuming more if they find that their estrogen levels are higher than what they should be. There are actually two different types of chemicals in red grapes that assist with the blocking of a process in the body that produces estrogens.
RAW CARROTS
Carrots actually contain unique undigestible fibres to help detox excess estrogen from the body. Raw carrots have been found to help by preventing the reabsorption of estrogen from the intestine, meaning the liver can more effectively regulate metabolism.
Genes associated with follicle growth were not significantly altered by vitamin D3. However, it increases expression of genes involved in the estrogen-biosynthesis. Further, estrogen concentrations in porcine granulosa cell-cultured media increased in response to vitamin D3.
Diet-related causes of elevated estrogen levels
Your overall diet can impact your hormone levels. Research shows that Western-type diets high in ultra-processed foods, added sugar, and animal foods (like red meat and eggs) are associated with high estrogen levels.
Another source of estrogen in foods is animal estrogen. Products like eggs or milk contain high estrogen levels because they are produced in parts of the animal's body that regulate its hormones. Eating high estrogen foods can help people who suffer from various conditions related to low estrogen levels.
Zinc can rebalance these hormone levels through its antiandrogenic activity which will raise estrogen and progesterone back to baseline.
The FDA has approved elacestrant (Orserdu) for the treatment of postmenopausal women or adult men with estrogen receptor–positive, HER2-negative, ESR1-mutated advanced or metastatic breast cancer with disease progression following at least 1 line of endocrine therapy.